"re-assessing deco profiles and deep stops", plus other bits..

What do you think you might see if we took a close look into most any other model at the minimum deco / fast setting (ZHL 100/100 for example).
So acute decompression is performed by only a few selected combat divers.
With the right options decompression chamber on board a warship.
For the rest of the population is an invitation to the cemetery.
Saturation limit value for the Bühlmann model is 0.9 M (h) for the start of decompression, it can be translated into 75-80%.

If you look at the mechanics of wound healing, you can see how the body can heal itself after an injury (possibly asymptomatic), but not necessarily in case of nerve damage (my understanding is that the body can find new pathways rewiring itself, but nerve damage does not "heal" and is irreversible hence "injury" is irreversible, although may be asymptomatic).

Formation of bubbles destroying nerve cells have a slightly different mechanism. There is no connection bloodstream of nervous tissue. Bubbles generated there is another problem. Here is the mechanism: the nerve cell axon is the myelin. Which was dissolved in a lot of nitrogen (oil soluble nitrogen five times more than the water)). With the rapid decompression (adipose tissue has a low saturation limit, lower than the tissue water) gas bubble is formed which is experiencing axon. Some cells are able to regenerate, most after this incident no longer transmit nerve signals.
Modeling of such processes are needed in series parallel models.

greet rc
 
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